[01] Samir Patel, Department of Neurology, Sir Ganga Ram Hospital, New Delhi, India. [02] Ish Anand, Department of Neurology, Sir Ganga Ram Hospital, New Delhi, India. [03] Anuradha Batra, Department of Neurology, Sir Ganga Ram Hospital, New Delhi, India. [04] Pooja Gupta, Department of Neurology, Sir Ganga Ram Hospital, New Delhi, India. [05] Rajat Agrawal, Department of Neurology, Sir Ganga Ram Hospital, New Delhi, India.

Guillain-Barre syndrome is an acute polyradiculoneuropathy, often triggered by a preceding bacterial or viral infection. Campylobacter jejuni, mycoplasma pneumoniae, cytomegalovirus and Epstein–Barr virus are the common triggering agents. Infectious agents induced molecular mimicry is supposed to be involved in the pathogenesis of GBS. Rarely, it has been observed in association with hepatotropic viruses (Hepatitis A, B, C and E). However, the nature of the shared epitopes has not been characterized in hepatotropic viruses. We report a case of Guillain-Barre syndrome following acute hepatitis A in a young Indian female.

Guillain-Barre Syndrome, Hepatitis A

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